Fearful memories can be rewritten.

Researchers at New York University have demonstrated scientifically that a specific fearful memory can be rewritten in the brain without the use of drugs – purely behaviorally. Of course, alternative practitioners like hypnotherapists and Rapid Eye Technicians have seen this over and over and are sold on the fact that fearful memories can be rewritten (in NLP it’s called “Reframing”).

Basing their theories on mouse and rat subjects, the researchers, led by Elizabeth Phelps, Ph.D., and Joseph LeDoux, Ph.D., of NYU, grantees of the National Institutes of Health’s National Institute of Mental Health (NIMH), have demonstrated their training process on human subjects with positive results. The hope is to replace drug therapies with behavioral ones for anxiety and PTSD specifically – and perhaps others after some trials.

The research shows that there is a critical window of opportunity for change – within 6 hours of the recall of a traumatic memory. Once the “file” is open, specific behavioral techniques can be used to rewrite the memory back into the brain without the fear portion – with long-lasting results. The researchers also found that it was not necessary to recall specifics within a memory – just the emotional elements and the “gist” of the traumatic memory – in order to rewrite it. That’s the phenomenological findings of thousands of Rapid Eye Technicians, who basically tell their clients, “It’s not necessary to relive the events in order to release their energy and reframe [rewrite] those memories…”

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This research strongly suggests that the emotional content of long-term memories can be removed by interrupting the labile phase of long-term memory storage.

Another study, this time from the Universiteit van Amsterdam, demonstrates that memories – most particularly long-term fear memories – are encoded when they first happen and then again whenever we re-store those memories. There is a short period of time in which the brain must chemically “prepare” and then “store” the memory. Whenever we bring the memory back to mind, it must go through the same process to re-store it in the brain. In both of these labile phases, the memory is vulnerable to change.

This research strongly suggests that memories are not, therefore, permanent structures in the brain. Their emotional content can be removed by interrupting the labile phase of long-term memory storage.

I wrote about the brain’s file cabinet in another post (Click here to read). Basically, the brain requires a chemical to access memories and to code them back after accessing them. It’s as though we take each memory, like a file, out of the long-term memory cabinet, close the cabinet, look at the file, use it, then open the cabinet again to put the file back in. If I understand Kindt’s research correctly, interrupting that process at the “putting back in” phase can, in theory, stop the perpetuation of the effects of fear memories by eliminating the fear in the memories.

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People who complain that they are more sensitive to sadness or frustration than others and report feeling “hurt” may be telling the truth.

There are approximately three billion base pairs (connections) in a strand of DNA. That represents a virtually infinite number of possible combinations. The variation between each of us, although nearly infinitesimally small, is so significant that no two of us in the world population of nearly 7 billion humans is exactly identical. Even identical twins are different from each other.

It is that small variation in each of us that is the result of and contributes to the evolution of the specie. In a study by UCLA researchers, publishing in Proceedings of the National Academy of Sciences, Markus Heilig, Faculty Member for F1000 Biology, and Chief of the Laboratory of Clinical and Translational Studies at the National Institute on Alcohol Abuse and Alcoholism, Bethesda, MD, report that they have identified a genetic factor that causes some people to actually experience stronger physical sensations associated with emotions (in the study case, the emotion was rejection).

There is apparently a wide variation or spectrum associated with the feeling (physical sensations) of emotions. Therefore, some people who complain that they are more sensitive to sadness or frustration, for example, than others and report feeling emotionally “hurt” may be telling the literal truth.

Can these genetic factors be moderated through training or experience? What do you think? Are we “hard wired” – or can we significantly affect our emotional states in spite of genetic predisposition or genetic variations?

An abstract of the original paper, Variation in the micro-opioid receptor gene (OPRM1) is associated with dispositional and neural sensitivity to social rejection is online at http://www.ncbi.nlm.nih.gov/sites/entrez/19706472?dopt=Abstract&holding=f1000,f1000m,isrctn.

Teenagers tend to pick up the “vibes” of their friends more strongly than do younger children or adults.

It is a well-studied and known phenomenon – teenagers pick up the “vibes” of their friends more strongly than do younger children or adults. During adolescence, we bond very closely to friends. We pick up on their hurts and joys, sharing them in a much more psychologically intimate way than at other times in our lives.

I believe we may also pick up our friends’ traumas and make them our own. More than once have I worked with a client reporting childhood, teen, or young adult trauma that later turned out to be “ghosts” – imaginings based on a friend’s childhood trauma introduced to the shared sensitivities of an intimate group of young friends.

In other words – a false memory. Still, a memory with all the power and influence of a real trauma. And I, as the clinician, treated the symptoms of that trauma as though the original trauma belonged to my client. My client “owned” it, so why not treat it as thought it belonged to my client? Made sense to me. The mind is unable to differentiate between real and imagined when it comes to trauma.

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CO2 is heavier than normal air and so will tend to sit at the bottom of your lungs unless you exhale it.

John Wemmie and Michael Welsh of the University of Iowa, Iowa City, reported in the November 25, 2009 issue of the journal Cell, a Cell Press publication, that they have discovered a chemical sensor for carbon dioxide deep in the brain’s emotional center – the amygdala. This part of the brain, when it senses an acidic condition (ph) created by higher levels of CO2 in the body, triggers fight-or-flight behaviors we label panic attacks.

I’ve discussed before in this blog the impact of chemistry, particularly CO2 levels in the body on mood and behavior. This is not a new concept – it’s been known for at least a century (the Chinese knew it millenia ago) that chemical imbalances in the body affect behavior.

If you’re experiencing panic attacks fairly often, it could be that your amygdala is hypersensitive to CO2 levels. You may need to breathe differently to expel excess CO2 and increase the oxygen levels in your body.

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